Research Nootropics Europe — RUO Reference Compounds | Condor Research

Condor Research supplies research-grade nootropic and cognitive-research compounds across Europe for in vitro / laboratory use only. Operated by Atrio Sciences s.r.o. in Slovakia, we test every batch through an independent third-party laboratory in the EU and issue a lot-specific Certificate of Analysis. This page maps the collection — neuropeptides, mitochondrial agents, NNMT inhibitors, neurotrophic small molecules, polyamines and exercise mimetics — and explains how each category is documented. Framed strictly for laboratory research, not for human or veterinary use.

This page is informational and written for laboratory research contexts only. It does not describe human or animal dosing and contains no therapeutic, diagnostic or consumption claims. All materials are supplied for research use only (RUO).

What are research nootropics?

In a laboratory context, “nootropic” is a functional category rather than a regulatory classification — it encompasses structurally diverse compounds studied for their interactions with neuronal signalling, mitochondrial function, synaptic plasticity and neuroprotective mechanisms.1 The category includes synthetic neuropeptides originally developed in Soviet and post-Soviet pharmacology (Semax, Selank), mitochondrially active small molecules (Methylene Blue), NAD-pathway precursors (NMN, NAD+), angiotensin IV analogs investigated in cognitive animal models (Dihexa), and second-generation neurogenic compounds (J-147, 9-Me-BC). What they share from a research-chemistry standpoint is a defined molecular structure, the ability to cross or act at the blood-brain barrier in model systems, and a literature footprint in preclinical research.

For a research supplier, the relevant question is not a mechanistic claim but the same one that applies to any reference compound: is the material what it claims to be, at a characterised purity, tied to a traceable lot? That is the starting point for any meaningful laboratory experiment.

Why source research nootropics from Condor Research in Europe?

The non-peptide nootropic space is particularly prone to undisclosed impurities and misrepresented structures, because unlike well-characterised peptides, small molecules in this space are often synthesised by a smaller number of contract manufacturers and documentation is inconsistently passed through the supply chain. Condor’s quality model addresses this at the compound level:

What you get How it is established
Independent third-party testing A laboratory in Czechia — separate from the seller — characterises each lot before it is offered.
HPLC purity ≥99% Identity and purity by HPLC; the specification is ≥99% for single-compound formats.
Mass-spectrometry identity MS confirms the observed molecular mass matches the stated structure for each compound.
Lot-specific COA A Certificate of Analysis carrying the batch number, traceable to the exact material received.
EU storage & dispatch Inventory held in our Slovakia warehouse and shipped within the EU under DAP (Incoterms 2020).
Transparent legal entity Atrio Sciences s.r.o., IČO 57 669 651 — a named, registered company with a verifiable address.

A compound’s research utility collapses if its identity is uncertain. Documentation is not supplementary; it is the product. See our complete method overview on Quality & third-party testing.

A reference compound is only as useful as the evidence around it. We treat the COA — independent, lot-specific, method-stated — as the specification, not a marketing accessory.

The research nootropics collection

The catalogue is organised by mechanism and chemical class. Each compound links to its product page, where the listing is intended to carry its own batch COA.

Neuropeptides — Semax & Selank

Semax (ACTH(4-7)-Pro-Gly-Pro) is a synthetic heptapeptide derived from the adrenocorticotropin sequence, studied in Russian neuropharmacology for its interactions with brain-derived neurotrophic factor expression and neuroinflammatory markers in rodent and cell models.2 It is registered in Russia and Ukraine as a pharmaceutical, making it one of the few nootropic peptides with regulatory history, though not approved elsewhere. Selank (Thr-Lys-Pro-Arg-Pro-Gly-Pro) is a synthetic heptapeptide analog of the endogenous immunomodulator tuftsin, examined in anxiolytic and cognitive research contexts, with a literature profile covering GABAergic interactions and stress-response pathways.3

Compound Format Mechanism studied in preclinical literature
Semax Lyophilised vial · RUO BDNF upregulation, neuroprotection and neuroinflammatory-marker modulation in rodent and cell models.2
Selank Lyophilised vial · RUO Tuftsin analog; studied for anxiolytic-like effects and cognitive-performance interactions in animal models.3
Semax + Selank blend Combination vial · RUO Co-administration blend; purity reported per component. See the Selank vs Semax research guide.

Further reading: What is Semax · What is Selank · Nootropic Peptides Explained.

Mitochondrial & energy-metabolism agents

This sub-category groups compounds studied for their interactions with mitochondrial electron-transport chains, NAD⁺ redox balance, and ATP synthesis in neuronal and non-neuronal model systems.

Methylene Blue (3,7-bis(dimethylamino)phenothiazin-5-ium; MB) is a phenothiazine dye with a well-characterised redox-cycling mechanism. In vitro and animal studies have examined its role as an alternative electron carrier in mitochondrial complex I–IV bypass, its effects on cytochrome c oxidase activity, and potential neuroprotective interactions.4 A rodent study observed attenuation of cognitive deficits associated with mitochondrial dysfunction in a surgical-anaesthesia model.5

NAD⁺ / NMN: Nicotinamide adenine dinucleotide (NAD⁺) is the central coenzyme of cellular redox metabolism and a substrate for sirtuins (SIRT1–7) and PARPs. Nicotinamide mononucleotide (NMN) is the immediate biosynthetic precursor to NAD⁺ via the NMN adenylyltransferase (NMNAT) pathway. Mills et al. observed that long-term oral NMN administration in aged mice mitigated physiological decline across multiple parameters including energy metabolism, bone density and eye function — outcomes attributed to NAD⁺ restoration.6

Compound Format Research context
Methylene Blue Capsules Encapsulated · RUO Mitochondrial electron-carrier and redox-cycling research; neuroprotection model studies.45
NAD+ Vial · RUO Redox coenzyme; sirtuin and PARP substrate research; cellular energy-metabolism studies.
NMN Capsules Encapsulated · RUO NAD⁺ precursor via NMNAT pathway; studied in metabolic and mitochondrial aging models.6

Further reading: What is NAD+ · What is NMN · NMN vs NAD+ comparison · What is Methylene Blue · NAD/NMN/NR: Human Evidence Review.

NNMT inhibitor — 5-Amino-1MQ

5-Amino-1-methylquinolinium (5-Amino-1MQ) is a selective small-molecule inhibitor of nicotinamide N-methyltransferase (NNMT), an enzyme that methylates nicotinamide (consuming S-adenosyl methionine in the process) and is upregulated in adipose and tumour tissue. NNMT inhibition in preclinical models has been studied in the context of metabolic regulation, fat-mass reduction and effects on NAD⁺ precursor availability, because blocking NNMT conserves the methyl-donor pool and funnels nicotinamide toward NAD⁺ synthesis.7

Compound Format Research context
5-Amino-1MQ Capsules Encapsulated · RUO Selective NNMT inhibitor; studied for metabolic and NAD⁺-pathway effects in cell and rodent models.7
5-Amino-1MQ (Vial) Lyophilised vial · RUO Same compound; vial format for researchers requiring solution-based application.

Further reading: What is 5-Amino-1MQ · 5-Amino-1MQ vs SLU-PP-332.

Neurotrophic small molecules — Dihexa, J-147, 9-Me-BC

This group covers non-peptide small molecules with neurotrophic or neurogenic profiles in preclinical literature. Dihexa (N-hexanoic-Tyr-Ile-(6) aminohexanoic amide) is a synthetic angiotensin IV analog designed to penetrate the blood-brain barrier and potentiate hepatocyte growth factor (HGF) / c-Met signalling, a pathway implicated in synaptic plasticity. In an APP/PS1 transgenic mouse model, Dihexa was reported to rescue cognitive impairment and restore memory via PI3K/AKT pathway activation.8

J-147 is a curcumin-derived compound developed at the Salk Institute. A 2013 aged Alzheimer’s disease mouse study reported cognitive-impairment reversal with J-147 treatment, attributed in part to enhancement of memory-associated gene expression and neurogenesis markers.9

9-Me-BC (9-methyl-β-carboline) is a β-carboline derivative studied for dopaminergic neuroprotective effects. In rodent neuronal models, 9-Me-BC was shown to stimulate dopaminergic neuron growth and upregulate expression of neurotrophic factors including GDNF and BDNF, suggesting a profile relevant to dopamine-pathway research.10 A separate in vivo study reported 9-Me-BC-induced cognitive enhancement correlated with elevated hippocampal dopamine levels.11

Compound Format Research context
Dihexa Capsules Encapsulated · RUO Angiotensin IV analog; HGF/c-Met potentiation and cognitive-rescue studies in transgenic mice.8
J-147 Capsules Encapsulated · RUO Curcumin derivative; neurogenesis and cognitive-impairment-reversal research in aged AD mouse models.9
9-Me-BC Capsules Encapsulated · RUO β-carboline derivative; dopaminergic neuroprotection and BDNF/GDNF upregulation in neuronal models.1011

Further reading: What is Dihexa · What is J-147 · What is 9-Me-BC.

Polyamines — Spermidine

Spermidine is a naturally occurring polyamine found in ribosomes, nuclear chromatin and cytoplasm across all kingdoms of life. The landmark Eisenberg et al. study (2009, Nature Cell Biology) demonstrated that exogenous spermidine extends lifespan in yeast, flies and worms by inducing autophagy — a mechanism dependent on inhibition of the acetyltransferase EP300 and subsequent histone hypoacetylation.12 Condor Research supplies the trihydrochloride salt form for aqueous solution-based laboratory studies.

Compound Format Research context
Spermidine Trihydrochloride Powder / vial · RUO Autophagy induction, polyamine biology, and longevity-pathway research in multiple model organisms.12

Further reading: Spermidine Autophagy Research Guide.

Exercise mimetics / metabolic — SLU-PP-332

SLU-PP-332 is a synthetic agonist of estrogen-related receptors (ERR-α, ERR-β, ERR-γ) — nuclear receptors that regulate mitochondrial biogenesis and oxidative metabolism programs, including pathways activated by endurance exercise (PGC-1α, OXPHOS gene clusters). It is studied as a tool compound for dissecting ERR-mediated transcriptional control of metabolism in cell and animal models, and has been described in the literature as an “exercise mimetic” due to these downstream metabolic effects.

Compound Format Research context
SLU-PP-332 Lyophilised vial · RUO Pan-ERR agonist; mitochondrial biogenesis and exercise-mimetic pathway research.
SLU-PP-332 Capsules Encapsulated · RUO Capsule format for study designs requiring pre-weighed single-unit application.

Further reading: What is SLU-PP-332 · 5-Amino-1MQ vs SLU-PP-332 · Exercise Mimetics Explained.

Browse the full nootropics catalogue

The compounds above represent the flagship lines; the full listing — including additional neuropeptides, bioregulators (Epitalon, Pinealon, Cortagen, Vesugen) and longevity compounds — is browseable by category:

Quality, purity and documentation

The ≥99% HPLC purity specification describes the chromatographic purity of the target compound under the laboratory’s method for that lot. Mass-spectrometry identity confirmation validates that the observed molecular mass matches the stated structure. These are analytical characterisations for laboratory research. They do not describe safety, efficacy or any application in humans or animals, and a COA is lot-specific — it speaks to the tested material, not a generic specification. Stating those limits is itself part of quality documentation. For the full method and what a COA contains, see Quality & third-party testing and How to Read a Certificate of Analysis.

EU logistics

Inventory ships from our warehouse in Nitra, Slovakia via Packeta, DPD or DHL, with free shipping on orders of €150 or more, under DAP (Incoterms 2020). Pricing is in EUR. These compounds are sold as B2B reference materials; sales are final except where mandatory law provides otherwise. EU-based storage and dispatch reduce transit time and customs friction relative to non-EU suppliers.

Frequently asked questions

Are research nootropics legal in Europe?

Reference compounds supplied for in vitro / laboratory research with appropriate documentation (SDS/CLP under REACH where applicable) and without any human or veterinary use claim are sold and used as research materials within the EU. The RUO frame must be genuine — not a disclaimer bolted to the footer. Legality is compound- and jurisdiction-specific; researchers remain responsible for compliance in their own country.

Do you test your nootropic compounds independently?

Yes. Every batch is characterised by an independent third-party laboratory in the EU (Czechia) before listing. HPLC establishes purity (≥99% for single-compound formats), and mass spectrometry confirms identity for each compound. Each batch ships with a lot-specific Certificate of Analysis traceable to the material you receive.

Which nootropic research compounds does Condor Research supply?

The current catalogue includes: neuropeptides (Semax, Selank, Semax+Selank blend); mitochondrial / energy agents (Methylene Blue, NAD+, NMN); NNMT inhibitors (5-Amino-1MQ capsules, 5-Amino-1MQ vial); neurotrophic small molecules (Dihexa, J-147, 9-Me-BC); polyamines (Spermidine Trihydrochloride); and exercise-mimetic / metabolic compounds (SLU-PP-332 vial, SLU-PP-332 capsules).

Do you ship nootropic compounds across the EU?

Yes. Inventory ships from our Slovakia warehouse via Packeta, DPD or DHL, under DAP (Incoterms 2020). Free shipping on orders of €150 or more. EU-based dispatch reduces customs friction compared with non-EU suppliers.

What does Research Use Only mean for these compounds?

It means the materials are reference compounds for laboratory and in vitro investigation only. They are not drugs, foods or supplements; they are not for human or veterinary use; and they are not for diagnostic or therapeutic purposes. No dosing or consumption guidance is published, because that would contradict the RUO frame.

Who is Condor Research?

Condor Research is operated by Atrio Sciences s.r.o., IČO 57 669 651, registered at Hornočermánska 1556/76, 949 01 Nitra, Slovakia, with EU-based testing and dispatch. Questions: info@condorresearch.com.

Start with the catalogue

Browse the full Nootropics & Cognitive category, or go directly to a flagship compound: Semax, Methylene Blue, NMN or Dihexa. Each listing is intended to carry its own batch documentation.

All products supplied by Condor Research (Atrio Sciences s.r.o.) are for research use only / in vitro use. They are not for human or veterinary use, not drugs, foods or supplements, and not for diagnostic or therapeutic purposes. Nothing on this page is dosing, medical or consumption advice.

Condor Research · Scientific desk

References

  1. Giurgea C. Vers une pharmacologie de l’activité intégrative du cerveau. Actual Pharmacol (Paris). 1972. [Foundational definition of “nootropic” — no PMID; primary literature.] Historical reference.
  2. Dolotov OV, et al. Brain Protein Expression Profile Confirms the Protective Effect of the ACTH((4-7))PGP Peptide (Semax) in a Model of Focal Ischemia in Rats. Int J Mol Sci. 2021 Jun 8. PMID: 34201112. https://pubmed.ncbi.nlm.nih.gov/34201112/
  3. Kolomin T, et al. Peptide-based Anxiolytics: The Molecular Aspects of Heptapeptide Selank Biological Activity. Protein Pept Lett. 2018. PMID: 30255741. https://pubmed.ncbi.nlm.nih.gov/30255741/
  4. Bhatt S, et al. From Mitochondrial Function to Neuroprotection — an Emerging Role for Methylene Blue. Mol Neurobiol. 2018 Jun. PMID: 28840449. https://pubmed.ncbi.nlm.nih.gov/28840449/
  5. Mei X, et al. Methylene Blue Protects Against Sevoflurane-Induced Cognitive Dysfunction by Suppressing Drp1 deSUMOylation in Rats. Neurochem Res. 2020 Apr. PMID: 32008150. https://pubmed.ncbi.nlm.nih.gov/32008150/
  6. Mills KF, et al. Long-Term Administration of Nicotinamide Mononucleotide Mitigates Age-Associated Physiological Decline in Mice. Cell Metab. 2016 Dec 13. PMID: 28068222. https://pubmed.ncbi.nlm.nih.gov/28068222/
  7. Kannt A, et al. Reduced calorie diet combined with NNMT inhibition establishes a distinct microbiome in DIO mice. Sci Rep. 2022 Jan 10. PMID: 35013352. https://pubmed.ncbi.nlm.nih.gov/35013352/
  8. Bose P, et al. AngIV-Analog Dihexa Rescues Cognitive Impairment and Recovers Memory in the APP/PS1 Mouse via the PI3K/AKT Signalling Pathway. Brain Sci. 2021 Nov 11. PMID: 34827486. https://pubmed.ncbi.nlm.nih.gov/34827486/
  9. Currais A, et al. The neurotrophic compound J147 reverses cognitive impairment in aged Alzheimer’s disease mice. Alzheimers Res Ther. 2013. PMID: 23673233. https://pubmed.ncbi.nlm.nih.gov/23673233/
  10. Polanski W, et al. Stimulation, protection and regeneration of dopaminergic neurons by 9-methyl-β-carboline: a new anti-Parkinson agent? Expert Rev Neurother. 2011 Jun. PMID: 21651332. https://pubmed.ncbi.nlm.nih.gov/21651332/
  11. Hamann J, et al. 9-Methyl-β-carboline-induced cognitive enhancement is associated with elevated hippocampal dopamine levels and dendritic and synaptic proliferation. J Neurochem. 2012 Jun. PMID: 22380576. https://pubmed.ncbi.nlm.nih.gov/22380576/
  12. Eisenberg T, et al. Induction of autophagy by spermidine promotes longevity. Nat Cell Biol. 2009 Nov. PMID: 19801973. https://pubmed.ncbi.nlm.nih.gov/19801973/